Researchers from Tel Aviv University and the Israel Institute for Biological Research in Ness Ziona have used the platform ...

The virus binds to that cell, slips inside and uses the cell's machinery to help make copies of itself. ... The T cells help B cells turn on to produce antibodies that could bind to the actual virus.

In the vast and often unseen world of microscopic life, a recent discovery may force scientists to rethink what it means to ...

A study published in mBio details the vulnerability of coronaviruses to inhibitors of a small protein domain called Mac1, or ...

It has been suggested, however, that the actual amino-acid sequence of the membrane-spanning domain is not critical for the activity of viral fusion proteins. We have examined the function of Moloney ...

As a professor of visual communications, I can’t help but notice all the images of COVID-19 that have been circulating. You’ve probably seen some version of it: a ...

The coronavirus SARS-CoV-2 may latch more easily onto the airway cells of people with type A blood compared with those with type B or O blood, a new study suggests.

After a virus invades a cell, it hijacks its machinery to make copies of itself. White blood cells known as cytotoxic T cells, a.k.a. killer T cells, can identify the wayward cell and make it self ...

Adeno-associated virus type 2 infection activates caspase dependent and independent apoptosis in multiple breast cancer lines but not in normal mammary epithelial cells. Molecular Cancer , 2011 ...

Later, immune cells use these proteins to produce antibodies that will block the actual virus' spike proteins from binding to people's cells and infecting them. How an anti-vaxxer's speculation ...

I’ve had it up to here with viruses lately. It’s easy in these times to wonder whether there’s anything good about them. That made me curious about why viruses exist in the first place, and ...