The virus binds to that cell, slips inside and uses the cell's machinery to help make copies of itself. ... The T cells help B cells turn on to produce antibodies that could bind to the actual virus.

New imaging tools reveal how within an hour of infection, the virus begins to alter our chromosomes to kick-start its own replication.

Scientists have identified dozens of human proteins that SARS-CoV-2, the virus that causes COVID-19, depends on to replicate ...

A study published in mBio details the vulnerability of coronaviruses to inhibitors of a small protein domain called Mac1, or ...

As a professor of visual communications, I can’t help but notice all the images of COVID-19 that have been circulating. You’ve probably seen some version of it: a ...

It has been suggested, however, that the actual amino-acid sequence of the membrane-spanning domain is not critical for the activity of viral fusion proteins. We have examined the function of Moloney ...

But these surrogates can’t tell you what the actual virus does to specific human cell types. “If you work with the real thing, you get real results,” says Elke Mühlberger, ...

The coronavirus SARS-CoV-2 may latch more easily onto the airway cells of people with type A blood compared with those with type B or O blood, a new study suggests.

Adeno-associated virus type 2 infection activates caspase dependent and independent apoptosis in multiple breast cancer lines but not in normal mammary epithelial cells. Molecular Cancer , 2011 ...

After a virus invades a cell, it hijacks its machinery to make copies of itself. White blood cells known as cytotoxic T cells, a.k.a. killer T cells, can identify the wayward cell and make it self ...

Using an approach the lab called PAC-MAN (Prophylactic Antiviral Crispr in huMAN cells), the idea was to attack the coronavirus by directing a Crispr torpedo at it, attacking the virus’s genetic ...